JК
Английский язык :: Беликова Елена
Страница: 62 из 68Each pyramid has a free rounded projection – a renal papilla – which lies in a cap – like extension, of the pelvis cal led a renal calyx. The pelvis is lined by transitioual epithelium, which extends the calyces and covers the papillae
Within the cortex each minute artery presents along its course a convoluted knot, called a glomerulus; the branch which enters the knot is the afferent vessel, that which leaves is she efferent vessel. Each glomerulus project into the dilated end of its corresponding renal tubule, from which it is separated by a thin layer of cells called glomerular (Bowman s) capsule; glomerulus plus capsule form a renal (Nalpighian) corpuscle. The cortex contains multitudes of such corpuscles, each giving rise to a tubule which passes down into the medul la and back again in the so-called loop of Henle. Back in, the cortex loop ends in a functional tubule which joins а larger collecting tube. Ultimately, a number of collecting tubes combine to form an excretory tube, which opens at the ареx of a papilla into a renal calyx. The efferent vessel from the glomerulus accompanies the loop of Henle, supply ing the tubule on the way and finally ends in a small vein. A renal corpuscule plus its complement of tubules and blood vessels is called a renal unit, or nephron; there are said to be one million such units in each kidney, their tubing totaling a length of some twenty miles.
New words
bean-shaped organ – орган в форме боба
four inches long – 4 дюйма в длину
two inches wide – 2 дюйма в ширину
peritoneum – брюшина
lumbar – поясничный
renal cortex – корковый слой
renal medulla – мозговой слой
fibrous – волокнистая
dilated – расширенный
to be separated – быть разделенным
loop of henle – петля Генле
52. Acute renal failure
The two major mechanisms may participate in association between intratubular hemorrage and nephron damage in acute renal failure. The first mechanism is direct nephrotoxicity from hemoglobin, because intratubular degradation of erythrocytes releases heme and iron which are toxic to cells. The second mechanism is hypoxic damage induced by regional vasoconstriction because heme avidly binds the potent vasodilator nitric oxide.
Intratubular degradation of hemoglobin releases heme containing molecules and eventually free iron.
Within the cortex each minute artery presents along its course a convoluted knot, called a glomerulus; the branch which enters the knot is the afferent vessel, that which leaves is she efferent vessel. Each glomerulus project into the dilated end of its corresponding renal tubule, from which it is separated by a thin layer of cells called glomerular (Bowman s) capsule; glomerulus plus capsule form a renal (Nalpighian) corpuscle. The cortex contains multitudes of such corpuscles, each giving rise to a tubule which passes down into the medul la and back again in the so-called loop of Henle. Back in, the cortex loop ends in a functional tubule which joins а larger collecting tube. Ultimately, a number of collecting tubes combine to form an excretory tube, which opens at the ареx of a papilla into a renal calyx. The efferent vessel from the glomerulus accompanies the loop of Henle, supply ing the tubule on the way and finally ends in a small vein. A renal corpuscule plus its complement of tubules and blood vessels is called a renal unit, or nephron; there are said to be one million such units in each kidney, their tubing totaling a length of some twenty miles.
New words
bean-shaped organ – орган в форме боба
four inches long – 4 дюйма в длину
two inches wide – 2 дюйма в ширину
peritoneum – брюшина
lumbar – поясничный
renal cortex – корковый слой
renal medulla – мозговой слой
fibrous – волокнистая
dilated – расширенный
to be separated – быть разделенным
loop of henle – петля Генле
52. Acute renal failure
The two major mechanisms may participate in association between intratubular hemorrage and nephron damage in acute renal failure. The first mechanism is direct nephrotoxicity from hemoglobin, because intratubular degradation of erythrocytes releases heme and iron which are toxic to cells. The second mechanism is hypoxic damage induced by regional vasoconstriction because heme avidly binds the potent vasodilator nitric oxide.
Intratubular degradation of hemoglobin releases heme containing molecules and eventually free iron.